Ringworm in humans symptoms
Ringworm - is a disease of an infectious nature, which affects the skin, mucous membranes rarely appears peeling, itching, burning sensation in the site of the lesion pathological agents.The mere word « zoster » just scares most people.
Ringworm means a dermatological disease that can occur for a variety of reasons, which differ, in turn, the appearance of spots on the skin itchy rash.Lisha differ in the nature of the rash spread and location.
The disease lasts a relatively long time.In some cases, there are periods of exacerbation and the risk of re-infection.In addition zoster it is always a discomfort with aesthetic inconvenience.
Like any other infectious disease, shingles can cause a variety of etiological factors.It is connected with this classification of lichen.So, shingles can be:
shearers or mikrosporiya;
- If detected ringworm, which is caused by the fungus Trichophyton, its manifestations affect the scalp.On it you can see
incubation period of ringworm takes five days from the moment of contact with pathogenic fungi for up to six weeks.Infected person is dangerous to others.
2. Pink zoster
- If you have a pink zoster, it is usually localized to the human body.At the same time on the site of the lesion occurs spot of pink color, at least - with a brownish tinge.Spot peripherally surrounded by a rim of red color, there is peeling.Spot appears first one, the so-called parent plaque, then another, subsidiaries plaques derived from the primary parent.When pink shingles affected the skin of the body: abdomen, back, shoulders, chest.Gibert
Ringworm in humans causes a variety of symptoms.frequent diseases falls on the period from age 20 to 40 years.As the disease progresses, you can feel the acute malaise, swollen lymph nodes, fever.On the skin appear symmetrical formation bright red or pinkish-yellow color.Usually education bulge above the skin surface for a few millimeters.
diameter spots of 1-2 cm. The sides of peeled skin spots, and in the middle of it is a little wrinkled.Along the perimeter there is a pinkish-red corolla.This rash spreads over the skin for 2-3 weeks, then gradually disappear, leaving behind a white or pink spots.Over time, the traces of the rash disappear.
In half the cases, before zoster appears on the body, a so-called "parent plaque" - a large stain of 3-4 cm in diameter with a bright pink color with a surface covered with pityriasis scales.
zoster most often appears on the chest, then goes down slowly in the stomach to the inguinal folds, spreading hips, shoulders and neck.Less often seen on the face.
When the manifestation of the disease possible occurrence of itching and temperature.After 4-5 weeks of pink spots begin to fade and disappear.
Pink zoster not typical forms
By atypical forms of expression may include depriving a bubble, drain or spot rash.As there is a ring-shaped lichen Vidal.The number of lesions is small, but each spot reaches 8cm.in diameter.It is this form can occur chronically and remain on the body for many years.
etiology of this disease is unknown till the end, but there is an assumption that a certain role in the development of this type of depriving viral agents play.
- If diagnosed with shingles, it says that the third type of dormant herpes virus that causes chickenpox primary, activated and gives the pathological progression of the disease.At the same time as denying affects the nervous system in the course of the nerve trunks of any localization.
When shingles occurs characteristic clinical picture: there are pain along the nerve trunks, itching, burning.After that develops Vesicular eruptions along the nerve trunk.Vesicle - a bump filled with liquid.With the vesicles burst over time, in their place formed a crust that later fall off, leaving no trace.
Shingles - a sporadic disease that occurs as a result of activation of latent varicella-zoster virus.
characterized by inflammation of the posterior roots of the spinal cord and intervertebral ganglia, as well as the occurrence of fever, general intoxication and vesicular exanthema in the course of the process involved in sensory nerves.
Etiologiya- varicella zoster virus (herpes simplex virus type 3).Ill persons previously undergone chickenpox.Ill persons usually elderly.The incidence ranges from 5 to 10 per 1,000 people aged 60-80 years.In some cases (about 2% of patients with normal immunity and 10% of the patients with immunodeficiencies) disease occurs again.When contacted without a history of early children with sick shingles they have a typical chicken pox.
Shingles often occurs in people who are exposed to various influences that weaken the immune system (the patients of leukemia, Hodgkin's disease, tumors who receive chemotherapy, long receiving corticosteroids and immuno-depressants, most often infection occurs in cases with acquired immunodeficiency syndrome).
are ill elderly persons due to age-related decrease of immune protection.As a result excited the latent varicella-zoster virus infection, which for decades remained in the body without causing any clinical manifestations.A necessary component of the activation of the viral infection is a characteristic ganglionevrit with damage to intervertebral ganglia (ganglia or cranial nerve) and dorsal root damage.The virus can bring to the process of autonomic ganglia and provoke meningoencephalitis.May be affected and the internal organs.Therefore, in a picture of herpes zoster, unlike chicken pox to the fore not so much epite-lyotropic, as signs of neurotropic virus.
incubation period lasts for many years with shingles (on the transfer of primary infection prior to activation).
Distinguish following clinical forms of the disease:
2) ear and eye;
3) gangrenosum (necrotic);
4) shingles with damage to the autonomic ganglia;
gangliokozhnaya most common form of the disease begins with acute fever, symptoms of intoxication and sharply expressed severe pain at the site of the planned rash.After 3-4 days (sometimes only after 10-12 days), there is a characteristic rash.Location of pain and a rash similar to nerve damage (usually intercostal) and has a surrounding character.Pain sometimes become unbearable, worse when no longer touch the skin, while cooling, movement.In place of the vesicular rash first appears infiltration and flushing of the skin, which then appear grouped blisters filled with clear and then cloudy contents.The vesicles dry out and become crusts.Sometimes the disease is accompanied by intoxication and neuralgic pains, rash absent.When a skin rash pain are usually less severe.
peculiar clinical signs are eye and ear shapes shingles.In the form of eye depriving affected trigeminal node (Gasser's ganglion) and rash located along the branches of the trigeminal nerve (on the mucous membranes of the eyes, nose, on the skin of the face).When the ear is drawn form a crank assembly process, and rashes appear on the pinna and around it, and may be in the external auditory canal.May develop paralysis of the facial nerve.Rash preceded by symptoms of intoxication and fever.Very
identified trigeminal neuralgia, which can last for several weeks.In the form of eye-specific viral keratitis occur, rarely iritis, glaucoma.
gangrenosum (necrotic) form of shingles usually develops in people with weakened immune systems.There deeper skin lesions with the formation of the subsequent scarring.
meningoencephalitic form stripping does not occur very often.The disease is characterized not mild, mortality is higher than 60%.This form begins with gangliokozhnyh appearances, often in the intercostal nerves, although it may be in the cervical region.In the future, there are signs of meningoencephalitis (ataxia, hallucinations, hemiplegia, meningeal symptoms, coma can occur).The time from the appearance of skin lesions before the development of encephalopathy varies from 2 days to 3 weeks.
Any of the above forms may be accompanied by autonomic ganglia lesions with the development of non-standard for shingles symptoms (vasomotor disturbances, Horner's syndrome, urinary retention, constipation or diarrhea).
depriving Complications: transverse myelitis accompanied by motor paralysis.
Herpes zoster in HIV-infected and other immunocompromised runs hard.The duration of the period of occurrence of rash increases to 1 week, crusts that cover blisters dry up no earlier than the third week of illness.Those most at risk of developing shingles tend to patients with Hodgkin's disease or lymphoma, approximately 40% of them can be a rash, common across the surface of the skin.In 5-10% of patients with disseminated skin lesions develop viral pneumonia, meningoencephalitis, hepatitis, and other serious complications.When deployed
clinical gangliokozhnyh forms of tinea diagnosis is not difficult.Errors often occur in the initial stage of the disease when there are symptoms of intoxication, fever and acute pain.In such cases, the wrongly diagnosed angina, pleurisy, pulmonary infarction, renal colic, acute appendicitis and other
differentiate from standard herpes, erysipelas, acute eczema.;generalized form of shingles - chicken pox.For laboratory studies finding virus diagnosis used in microscopy or by immunofluorescence method, virus isolation in tissue culture, serological methods.
first disease event held days that aim to combat intoxication, pain relief and the prevention of generalization of infection.Independently treat shingles can lead to post-herpetic neuralgia.
4. Pityriasis versicolor
- In the presence of tinea versicolor affected skin.This can be seen colorless scaly patches.
Lichen pityriasis (multicolored shingles) - a fungal disease of the skin.
pathogen - fungi of the genus Malassezia (old name Pityrosporum orbiculare) living in the horny layer of the epidermis.The disease is prevalent in tropical countries, but in our climate affects 5-10% of people.Predisposing factors include increased sweating, seborrheic diathesis.Colorful lichen usually develops in people with excessive sweating, characterized by an explosion in the hot season.
in modern dermatological practice is one of the most important places occupied by fungal skin lesions.So according to foreign authors, the frequency of the disease is a multi-colored lichen in temperate countries - 2%, in tropical and subtropical to - 40%.Colorful lichen affects persons of different sexes.A comparison between the sick women and men is 2: 1.The disease is prevalent in young adults, the disease peak in 18 - 25 years.In 1846 Eyhshtedtom was the first time described the causative agent of multi-colored lichen.
pathogen transmission from patient multicolored lichen or carrier, for example, a total bed, or through shared with him clothing or linen, in principle, likely.However, most people are carriers of the same fungi of Malassezia, located in the skin (in areas rich in sebaceous glands) and do not cause disease.Therefore, multi-colored lichen is not a contagious disease.Chromophytosis begins, usually with vozninoveniya small, not very irritable and not overlooking the pink stain the skin surface.When
of infection pityriasis or skin colored lichen on the chest, back, neck, shoulder girdle, and rarely the scalp there are small (3-5 mm diameter) noninflammatory yellowish-brown spots with clear boundaries not quite smooth, with poskablivanii which revealed a slight defurfuration.As a result, the growth of peripheral spots become larger in size and are connected to major centers of the so-called geographical outlines.Subjective feelings are absent.For diagnostic use iodine sample Balser: stains smeared with tincture of iodine, then they are painted in dark brown, then wipe with alcohol: loosened fungus horny layer quickly absorbs iodine and stains tinea versicolor stand out sharply, turning into a dark brown color on the background of slightly yellowed intactskin.Do not try to put this trial alone.Under the influence of ultraviolet rays (particularly at sunburn) as a result of peeling on the ground of former eruptions are nezagoreshie-psevdoleykoderma spots.
Under Wood's lamp multi-colored lichen stains have a yellow glow.When
obsedovanii under a microscope doctor can see the characteristic picture - accumulation of the fungus filaments with rounded cells.Diagnosis is based on characteristic clinical symptoms and positive iodine sample.In problematic cases a microscopic examination of skin scales to detect
pathogen differential diagnosis is carried out in a number of cases with syphilitic roseola, which does not peel off, not combined in the solid lesions, iodine test with negative, and serological tests for syphilis are positive, there may be other manifestations of syphilis.Psevdoleykodermu must be differentiated from the true syphilitic leukoderma, where the small rounded (0.5-1 cm) or merle hypopigmented spots without clear boundaries are on a bit of pigmented skin posterolateral surfaces of the neck, sometimes razrostayas on the skin of the back;positive serological tests for syphilis and other features make it possible to distinguish it from psevdoleykodermy.
Against tanned skin spots look a little brighter.Spots susceptible to merge with the emergence of large pockets, but can exist in isolation.Inflammatory phenomena are absent, there is little defurfuration.
5. Lichen planus
- If the patient is infected with lichen planus, that it is characterized by complaints of intolerable itching in the affected area, the appearance of red bumps on the skin or mucous membranes.Subsequently, bumps merge to form plaques.
During lichen planus rash begins to appear in parts of the body such as the chest, abdomen, arms and legs.The distinguishing feature - a spot in the form of a small nodule with the middle indentation.Ringworm
red flat - a disease that affects the skin, mucous membranes, nails less.
etiology, pathogenesis is not completely defined.There are neurogenic, viral and infectious-allergic theory of onset, attaches great importance to foci of chronic infection.There are also cases of lichen planus as an allergic reaction to some medication drugs (antibiotics, anti-malarial agents, etc..).In some patients decreased functional activity of the liver.Most adults fall ill, rare cases of disease of children.
clinical picture (symptoms of zoster) is characterized by monomorphic small polygonal papules reddish-purple color with a flat shiny surface and umbilicated in the center.Almost always it is possible to identify nodules in the skin of red color, with a smooth surface, pupkoobraznym depression, transverse striations.