The truth about accommodation

August 12, 2017 17:50 | Good Vision

my data esperimenty proved to me that the lens is not a factor in accommodation.This is confirmed by numerous studies eyes of adults and children both with normal vision, and with refractive errors, amblyopia (visual impairment to non-obvious cause), as well as studies of adults with a remote eye because of a cataract lens.We have already said that the instillation of atropine into the eye is intended to prevent accommodation by paralyzing the muscles responsible for the control of the shape of the lens.What it does produce such an effect may be in every textbook of ophthalmology and because atropine routinely used in the selection of points to exclude the estimated impact of the lens on the refractive state of the eye.

Somewhere in 9 cases out of 10 the state obtained as a result of instillation of atropine into the eye, with the theory on which his application is based.But in these cases, the tenth state obtained as a result of atropine does not correspond to its theoretical basis.Every ophthalmologis

t knows from experience that there are such cases.Many of them have been described in the literature, and I met during my own observations.According to the theory, atropine should identify latent hyperopia and apparently normal or explicitly hyperopic eyes, provided of course that the patient is at an age when the lens is supposed to be, still retains its elasticity.However, it is known that atropinizatsiya sometimes causes myopia or hyperopia converts in myopia.In people older than 70 years, when the lens is supposed to be tough as a rock (as in the case with early cataracts, when the lens is also cruel) atropine can cause both myopia and hyperopia.In patients with apparently normal eyes after using atropine developed hyperopic, compound myopic or mixed astigmatism.In other cases, the drug does not interfere with the accommodation or, in any case, a change in refraction.Moreover, when the vision was impaired by atropine, patients are often managed by giving a rest to the eyes, to read diamond type (usually the smallest size font to use, now known as the small print in 4 1/2 points) with 6 inches.However, it is believed that giving atropine eyes rest, removing the load from the overworked muscles.

the treatment of strabismus and amblyopia I have often applied atropine more than a year in the best eye to encourage use of amblyopic eyes.By the end of this period, is still under the influence of atropine, such eyes become capable of a few hours or less in order to read diamond type at six inches.The following are examples of many such histories.

The boy of ten years was hyperopia in both eyes.The left (the best) eyes had 3 diopters.When this eye-Ground atropine, hyperopia increased to 4.5 diopters, and decreased vision to 20/200 (200/200 - is the norm, the numerator is the distance from which the patient is able to see the letter on the test, and the denominator is the distancefrom which he had to see her, if he had normal vision).With convex lens 4.5 diopters the patient found a normal distance vision, and with the addition of another convex lens 4 diopter he was able to read diamond type 10 inches.Atropine is used throughout the year, the pupil dilates again and again to the maximum.Meanwhile, the right eye was treated by my own methods, which will be described later.Usually in such cases the eye sight, which is not treated in a special way, is improved to some extent, with the other eye sight, but in this case it did not happen.By year end, the right eye vision was normal, and the visual acuity of the left eye remained the same, exactly the same as it was in the beginning, making 20/200 without glasses for distance.In this vision of the left eye to read them without glasses it was not possible, because the degree of hyperopia has not changed.Still under the influence of atropine, with a pupil, extended to the maximum, the eyes are now treated separately.Just half an hour later his vision became normal both near and afar.Diamond type was read with a 6-inch glasses-free.According to generally accepted theories that the ciliary muscle of the eye should not only be at this time completely paralyzed, but also be in a state of complete paralysis of the whole year.However, this eye not only overcame the 4.5 diopters of hyperopia, but added six diopters of accommodation, making in total 10.5 diopters.We can only ask those who adhere to the generally accepted theories of how these facts are consistent with them.

equally, if not more remarkable, was the history of the disease a little six-year-girl, Right (the best) which the eye had 2.5 diopters of hyperopia, and the other - 6 diopters of hyperopia with astigmatism of one diopter.With the best eye, under the influence of atropine and the pupil, extended to the maximum, both eyes were treated with more than one year.By the end of this period (when the right eye was still on the influence of atropine), both eyes were able to read diamond type 6 inches, and the right eye is made, in any case, better than the left.Thus, in spite of the atropine, the right eye not only overcame the 2.5 diopters of hyperopia, but added six diopters of accommodation, making a total of 8.5 diopters.In order to exclude any possibility of latent hypermetropia in the left eye, which was originally 6 diopters, atropine steel now used therein, and the use of atropine in the other eye was discontinued.Coaching eyes continued as before.Under the influence of drugs was a slight return to hyperopia, but the vision quickly became normal again and while atropine is used on a daily basis for over a year, and the pupil again and again extended to the limit, diamond type was read from a distance of 6 inches without glasses during this period.I find it hard to understand how the ciliary muscle of the patient exercised accommodation, being under the influence of atropine year and more in each eye separately.

According to conventional theory, as I have said, atropine paralyzes the ciliary muscle and thus preventing the changing curvature of the lens, prevents the implementation of accommodation.Consequently, when the long-term use of atropine after a process of accommodation, it is obvious that this is made possible by another factor or factors than the lens and the ciliary muscle.Evidence given to the history, against the accepted theories are undeniable.Equally, these theories do not explain other steps in this article phenomenon.All these facts, however, are fully consistent with the results of my experiments on the muscles of animal eyes and research behavior of images reflected from various parts of the globe.They also confirm the excellent results of experiments with atropine, which showed that the accommodation is not prevented completely and permanently, if not deeply injected atropine in the eye socket so as to achieve the oblique muscles are real accommodation muscles.At the same time hypermetropia could not be prevented when the eyeball stimulated electric current without a similar use of atropine, leading to paralysis of the rectus muscles (see. Figure).

eye muscle

is well known that after the removal of the lens due to cataract, the eye is often able to accommodate the same way as before the operation.In their studies, I have seen many such cases.Patients in this case not only read diamond type with their glasses for distance from a distance of 13, 10, and less than an inch (the most difficult to read at a very small distance), but one patient was able to do it without any points.In all cases the retinoscope showed that there is a real accommodation, carried out not by some of the intricate ways that are usually explained by this "inconvenient" phenomenon, and the exact fit to the respective focus distances.

Clinical experiments directed against the generally accepted theory of accommodation, can be attributed to the elimination of presbyopia.According to that theory, where the lens is considered a factor in accommodation, such a change would be impossible.What the eye rest improves vision in presbyopia, and it was noted by other doctors.This is explained by the ability of the ciliary muscle relaxed for a short time to work on the hardened lens.This can prevent the early stages of presbyopia, and then for a short period of time.But it is impossible to assume that in this way a permanent positive effect and that hard as stone lens can be obtained, can succumb to any, even short-term exposure.

True enhanced accumulation of facts.The working hypothesis can not be considered true if it does not match any fact.The generally accepted theory of accommodation and causes refractive errors get off on a variety of facts superficial explanations.With more than thirty years of clinical experience, I have never seen a case contrary to the assertion that the lens and ciliary muscle have no relation to the accommodation and that changes in the shape of the eyeball, which depend on the refractive error, is not immutable.My clinical studies themselves are sufficient to demonstrate the truth of this assertion.They also are sufficient to show how you can optionally cause refractive errors and how they can be removed temporarily for a few minutes and always after long-term treatment.